Lymphedema People Logo


Recently on one of our lists, we had someone with questions regarding chronic venous insufficiency. Does it or can it cause edema? Yes, it can. Below are some selected sites that will give insights into this condition.

Related Terms:  Edema, lymphedema, post phlebitic syndrome, post-thrombitic syndrome, CVI, venous pooling, phlebo-lymphedema, lipodermatosis, ilio femoral disease, venous disease, diabetes militus


Chronic venous insufficiency is a condition of poor blood return from the lower extremities (feet, and legs) to the heart.

Risk Factors, Etiology:

Age, family history of deep venous thrombosis, sedentary lifestyle, obesity, smoking, deep vein thrombosis, congestive heart failure, diabetes mellitus, occupations that require long term standing.


Chronic foot or leg swelling in the affected limb, varicose veins, no healing leg ulcers, affected limb may experience pain pressure, itching, dull ache, or heaviness in the affected limb.  Skin changes which may include lipodermatosis, fat necrosis, fibrosis of the skin and subcutaneous layers. The skin color may become reddish or brown due to the accumulation of red blood cells.


Edema of the affected leg or foot, ulcerations, deep venous thrombosis, pigmentation and pain.


Radiological test may be prescribed to verify chronic venous insufficiency.  These test may include doppler bi-directional-flow studies, photoplethysmography , outflow plethysmography tests.  Other test may include venograms, and duplex ultrasounds.


Treatments will focus on two facets.  First the complications of CVI must be treated.  This may include decongestive therapy for the edema or swelling.  This may include not only decongestive massage and hosiery but compression pump therapy as well. Secondly, treatments will focus on the original cause of the CVI.

These treatments are broken into two categories.  First are the non-surgical treatments.  These will include leg elevation, compression stockings, use of Unna boots for chronic ulcerations, and injection sclerotherapy.

Surgical intervention will commonly be prescribed for patients who have CVI resulting from the congenital anomaly of weakened or non existent vein valves.  In this procedure, competent veins will be graft in replacement of defective veins.

For iliofemoral disease , the  operation of choice is a saphenous vein cross over graft.  In the procedure, the contralateral vein is mobilized and divided at its distal end. It is then tunneled suprapubilcally  and anastomosed to the femoral vein on the deceased side. The result is the diversion of venous blood through the graft  and into the intact contralateral venous system. (1)

Other treatments will focus on skin care to relieve itching, and ulcerations. Antibiotic therapy may be required for any infections resulting from the ulcerations.

Long term treatment will naturally focus on the underlying cause of the chronic venous insufficiency.

Diuretics may also be prescribed for a short term therapy.


Lymphedema and Chronic Venous Insufficiency


                     Chronic Venous Insufficiency              Lymphedema

Pain:              Yes                                                                  Can be severe due to nerve compression

Swelling:       Yes, may even include brawny edema.      Yes, can affect all or part of limb  

Infections:     Yes, from ulcerations                                   Yes, typically cellulitis, see below

Fluids:            Hematolgic Fluids                                        Protein rich lymphatic fluids

Skin:               Discolorations                                              Discolorations, growths, hardening

Leg Ulcers:   Yes                                                                 Yes, also wounds from skin changes

Fibrosis:        No                                                                   Yes, extending into subcutaneous tissues

Treatment for swelling from chronic venous insufficiency:

Manual decongestive therapy  (Complex or Complete Decongestive Therapy) is often prescribed.  Follow up by usage of compression garments. Compression bandages such as short-stretch bandages and long-stretch may also be used. Treatment will also focus on the cause of the chronic venous insufficiency

Treatment for swelling from lymphedema:

Manual decongestive therapy ((Complex or Complete Decongestive Therapy) is the gold standard treatment.  After decongestive therapy, compression bandages, custom fitted compression garments, compression sleeves,  are used.  Other treatment modalities may include the use of compression pumps, and surgery.  Treatment focus will be on the control and management of lymphedema not the cause.  There is no known cure for the cause of lymphedema.

Infections and leg ulcers from chronic venous insufficiency:

Chronic leg ulcers are a serious complication of chronic venous insufficiency.  Because of the insufficient blood, they are difficult to treat and may become septic foci for serious infections.  These infections may become gangrenous and are potentially life threatening. However with prompt and intensive antibiotic therapy, the infections will usually be resolved. Because the wounds are going to be difficult to heal, it may be necessary for the patient to seek treatment at a wound care clinic. For information on wound care see: How to Treat a Lymphedema Wound.

Infections related to lymphedema: 

Remember that In lymphedema, the effected limb is immunodeficient or immunocompromised so in both conditions, the patient is at an increased risk for infections.  Visual inspection of limbs is important. But, also learn the early warning signs of infections that at the very earliest signs antibiotic treatment can begin.  Also, with lymphedema patients it should be standard protocol to have an emergency bottle of antibiotics, prescribed by their physician on hand.

Early warnings signs of infections (cellulitis, lymphangitis) include all over body aching (much like the flu), excess and unexplained energy drain and or unexplained lethargy, susceptibility and sensitive to cold, increased urination, unusual sharp pain in the lymphedema limb, any sudden appearance of red spots, streaks, rash like areas or blotches. Besides cellulitis and  lymphangitis, the third significant infection we get is erysipelas.  You can get a overall rundown on bacterial infections by reading our page Infections Associated with Lymphedema and on fungal infections from our page Fungal Infections Associated with Lymphedema. You might also want to read our page Lymphedema Antibiotics, so that should you get an infection, you'll be informed on what antibiotics the doctors might use


Chronic Venous Insufficiency

What is it?

Chronic venous insufficiency (CVI) is a condition where blood pools in the veins of the lower legs.

Who gets it?

People who are obese, extremely inactive, or elderly may develop chronic venous insufficiency because all three factors can lead to problems with the valves in the veins. However, this condition can also be inherited.

What causes it?

The veins return blood to the heart from all the body’s organs. To reach the heart, the blood needs to flow upward from the veins in the legs. To do this, the calf muscles and the muscles in the feet need to contract with each step to squeeze the veins and push the blood upward. To keep the blood flowing up, and not back down, the veins contain one-way valves. Chronic venous insufficiency occurs when these valves become damaged, allowing the blood to leak backward. Varicose veins can damage the valves. Or, damage can occur when there is a problem with the calf muscle, so the blood isn’t being pushed upward. Chronic venous insufficiency can also be caused by a blockage in a vein, such as a blood clot. When any of these conditions occur, the blood can pool (called stasis) in the veins.

What are the symptoms?

The first signs of chronic venous insufficiency are ankle and leg swelling. Swelling occurs because the blood that has pooled in the veins causes abnormally high pressure in the veins. As the pressure and swelling increases, the skin of the legs may actually leak tiny drops of plasma, which is the pale yellow fluid part of blood. Eventually, the capillaries burst under the high pressure, releasing red blood cells and giving that area of the skin a reddish-brown discoloration. The discolored skin is easily broken by a scratch or bump. When this happens, the patient frequently develops leg ulcers, called venous stasis ulcers, which can become infected. An infected skin ulcer will ooze pus and have a foul-smelling discharge. If the infection spreads to the surrounding tissue, the patient develops a condition called cellulitis. Other symptoms of chronic venous insufficiency include legs that ache, feel heavy, or feel tired, especially after long periods of standing; new varicose veins; leg skin that looks and feels leathery; and flaking and itching in the affected area of the legs.

How is it diagnosed?

To diagnose chronic venous insufficiency, your doctor will take your medical history and carefully examine your legs. He or she may use a procedure called duplex scanning, which is a painless ultrasound examination of the leg veins.

What is the treatment?

Treatment for chronic venous insufficiency usually includes compression stockings, which squeeze the veins and keep blood flowing so it is more difficult for blood clots to form. If the skin is not broken or leaking fluid, your doctor may recommend an anti-itch cream, such as one containing hydrocortisone; a cream containing zinc oxide to protect the skin; or an antifungus cream to prevent fungal infections. Skin that is leaking fluid is treated with wet compresses. If you have ulcers on your legs, your doctor will show you how to apply layered compression bandages to protect the skin and maintain blood flow. Infected ulcers and cellulitis must be treated with antibiotics.

Self-care tips

If you have been diagnosed with chronic venous insufficiency, you can help the pooled blood to drain by elevating your feet whenever you are sitting or lying down. Your feet should be raised above the level of your heart. Avoid any long periods of sitting or standing. If you must take a long trip, flex and extend your legs, feet, and ankles about 10 times every 30 minutes to keep the blood flowing in the leg veins. If you wear elastic stockings, be sure to take them off each day to wash and dry them, and to clean and check your skin and give it some air. Make sure the stockings fit so there is no bunching. Elastic stockings that fit poorly will actually make your condition worse by blocking blood flow in the area where they have bunched up. Also keep your skin moisturized so that it doesn’t flake or crack easily. Follow your doctor’s recommendations for daily exercise so it is easier to maintain a healthy weight.



Venous insufficiency and ulceration: a review.
(Tips from Other Journals)

Alguire and Mathes conducted a MEDLINE search to review the current state of knowledge and treatment of chronic venous insufficiency and ulceration.

Venous valves control the flow of blood from the superficial veins to the deep veins in a distal to proximal direction. Incompetent valves allow backflow of blood when the muscles of the leg relax, contributing to venous pressures that are higher than normal. This venous hypertension is a major factor in chronic venous insufficiency. A patient with chronic venous insufficiency will typically present with varicose veins, tan or reddish brown changes in skin color and weeping, excoriated skin. These symptoms can progress to lipodermatosclerosis, the development of induration at the medial ankle or even to the mid-leg area. Ultimately, a brawny edema above and below the area of fibrosis can be seen. Ulcerations may develop in the fibrotic areas.

Venous stasis ulcers are more common in older women. These ulcers are chronic and frequently recurrent. Postphlebitic syndrome is the combination of chronic leg edema with deep venous thrombosis, pigmentation and ulceration.

Diagnosis is achieved by duplex ultrasonography (both B-mode and directional pulsed Doppler). Descending venography does not correlate as well as duplex scanning with the amount of venous reflux. Since the treatment for venous ulcers is not appropriate in cases of arterial insufficiency, the latter must be ruled out. One way of screening for arterial insufficiency is with the ratio of ankle blood pressure to brachial blood pressure (ankle/brachial index), which is also measured with Doppler ultrasonography. A normal score is greater than or equal to 0.9, claudication is indicated by a score of 0.5 to 0.9, and patients with resting ischemic pain usually score less than 0.5.

Treatment of chronic venous insufficiency consists of elevating the legs above heart level for at least 30 minutes three or four times daily, using compression stockings and using wet or dry nonadherent dressings or bandages. Compression stockings should apply a gradually decreasing amount of pressure from the ankle to the knee and should be applied on awakening. Some stockings have zippered backs or Velcro closures, making them easier to apply. For obese patients or those with a great deal of edema, intermittent pneumatic compression pumps may be used, although the pumps should not be used in patients with uncompensated congestive heart failure. Studies of various dressings have shown no significant difference in the rate of healing of venous ulcers, although patients seem to prefer the occlusive-type dressings because of their convenience.

The authors conclude that severe edema occasionally may require treatment with short-term diuretics. Topical antibiotics have not been shown to improve healing of the ulcer, although systemic antibiotics may be required for clearly infected ulcers or for cellulitis. Topical antiseptics, such as povidone-iodine, should be avoided because of cellular toxicity. The effectiveness of enzymatic debriding agents has not been proved. Some studies advocate the use of silver sulfadiazine, but other studies have shown no improvement in healing of the venous ulcers. Surgery has a very limited role in the treatment of chronic venous insufficiency.



Alguire PC, Mathes BM. Chronic venous insufficiency and venous ulceration. J Gen Intern Med 1997;12:374-83.

COPYRIGHT 1997 American Academy of Family Physicians

COPYRIGHT 1998 Information Access Company

This article has been provided courtesy of  Ames Walker Hosiery ( and may be reproduced for personal use provided no part of this article (including the text contents) has been changed. Copyright © 2003  Ames Walker International Inc.       


Horse Chestnut Seed Extract for
Chronic Venous Insufficiency

October 1999

Web_Cbooras_computer.gif (8591 bytes)

by Charles H. Booras, MD

Chronic venous insufficiency (CVI) describes persistent incompetence of the deep and perforating veins in the lower extremities. Swelling, darkening of the skin, and ulcerations can accompany leg pain, tiredness, and itching. CVI prevalence ranges from 10-15% in men and 20-25% in women. Traditional treatment includes external compression (support stockings) and for some, vascular surgery (venous stripping or sclerotherapy).

Extracts of horse chestnut seeds (HCSE) have been extensively studied and used for the treatment of CVI. The (German) Commission E has extensively evaluated and approved the use of HCSE for the treatment of CVI, especially when used as an adjunct to compressive therapy. There is good data to support HCSE’s effectiveness.


The medicinal portion comes from the dried leaves and seeds. The most active ingredients are called escins (or “aescins”). The (a)escin fraction, measured as a percent and reported in milligrams, has been standardized by some pharmaceutical manufacturers.

Mechanism of Action

In general, (a)escins are thought to act as anti-exudative and anti-inflammatory agents, decreasing vascular permeability and thus preventing edema formation.

Clinical Trials

To date, there are approximately 18 published randomized controlled clinical trials (RCTs) evaluating the safety and effectiveness of HCSE in patients with CVI. These studies have all been performed and published in Germany or France between 1973 and 1996.

In comparison to support stockings (compressive therapy), compliance with HCSE was better (98%) than with compressive therapy (90%). While the compression group achieved a peak effect at four weeks, the HCSE group achieved a similar effect at 12 weeks, but both therapies were clearly superior to placebo.

Adverse Effects and Drug Interactions

Oral HCSE appears to be well-tolerated. The most common side effects are itching, nausea, heartburn, headache, and dizziness. Most studies show no difference when compared to placebo. A recent observational study found that adverse events occurred in less than 1% of 5,000 patients treated with HCSE in therapeutic doses. There are no studies on the use of HCSE in pregnancy or while nursing, and no reported drug-drug interactions. However, there are two reports of kidney toxicity and one report of liver injury in patients given large doses of (a)escins. It is possible that HCSE may increase the blood-thinning activity of Coumadin®.

Formulation and Dosage
When used at the recommended dose, HCSE appears to be safe, effective and compares favorably to other currently available therapies for chronic venous insufficiency in the United States.

Oral Standardized HCSE preparations vary in their total milligram dosage which are usually expressed as a percent (from 16-22%) of (a)escins. The recommended dose is 50 mg (a)escins twice a day, with a maximum daily dose of 150 mg per day. Occasionally, HCSE is a component of a multi-herbal preparation that often includes Ruscus aculeatus or butcher’s broom, for which there are few good clinical data.

Until recently, standardized HCSE preparations were only available in Europe. In 1998, Pharmaton, a division of Boehringer Ingelheim Pharmaceuticals, released Venastat® in the United States for the “promotion of leg vein health.” This standardized HCSE represents one of the first examples of large pharmaceutical companies producing herbal preparations.


Standardized HCSE appears to be an effective treatment for the edema and symptoms associated with CVI and should be considered in the treatment of non-pregnant, non-lactating patients. While compression stockings are still considered first-line therapy, HCSE can be used alone in patients who cannot tolerate or comply with compressive therapy. The most effective regimen combines standardized HCSE with compressive therapy.

Because of the lack of data, HCSE should not be used in patients with acute or chronic DVT (blood clots in the deep veins of the legs), in patients on the blood-thinner Coumadin®, or in patients with kidney or liver insufficiency. Longer clinical trials in patients with similar grade CVI, using validated symptom measures and standardized leg volume measurements are still needed prior to HCSE becoming part of standard medical formularies.


Abstracts and Studies


Atypical skin lesions of the lower limbs in patients with chronic venous insufficiency: A case of granulomatous fungoid mycosis. 

Dec. 2011

[Article in French]
Labau D, Frouin E, Dereure O, Khau Van Kien A, Laroche JP, Guillot B, Quéré I.


Service de médecine interne et maladies vasculaires, hôpital Saint-Eloi, CHU de Montpellier, 34000 Montpellier, France.


In vascular medicine, venous insufficiency, ocre dermatitis, stasis dermatitis, or lipodermatosclerosis (level C4 in CEAP) may lead to skin lesions involving the lower limbs. Generally, symptoms resolve with etiologic treatment using medical compression, varicosis treatment, or dermocorticoids. However, some skin lesions progress, suggesting another diagnosis, including a specific dermatosis. The diagnosis is based on clinical, biological, radiological and histological criteria. Referral to a dermatologist may be necessary to determine the appropriate etiological treatment.


Acroangiodermatitis (pseudo-Kaposi sarcoma): a rarely-recognized condition. A case on the plantar aspect of the foot associated with chronic venous insufficiency

July !ug 2011

[Article in English, Portuguese]
Pimentel MI, Cuzzi T, de Azeredo-Coutinho RB, Vasconcellos Éde C, Benzi TS, de Carvalho LM.


Laboratório de Vigilância em Leishmanioses, Instituto de Pesquisa Clínica Evandro Chagas, Fundação Oswaldo Cruz, Rio de Janeiro, RJ, Brasil.


Acroangiodermatitis, often known as pseudo-Kaposi sarcoma, is an uncommon angioproliferative entity related to chronicvenous insufficiency, arteriovenous fistulae, paralysed limbs, amputation stumps, vascular syndromes and conditions associated with thrombosis. It presents most frequently as purple macules, papules or plaques in the dorsal aspects of the feet, especially the toes, and the malleoli. We report a case of acroangiodermatitis in the plantar aspect of the foot, misdiagnosed for two years, in which haematoxylin-eosin hystopathological stain and immunolabeling with CD34 histochemistry examination were decisive for diagnosis. Patient had chronic venous insufficiency. The lesion responded well to the treatment with a combination of leg elevation and compression.


Randomized Trial of Diosmin in Patients with Chronic Venous Insufficieny Following Coronary Artery Bypass Surgery and Deep

Venous Thrombosis

Arvind Kaul, Sandeep Seth, A Srivastava, SC Manchanda

All Institute of Medical Sciences, New Delhi

Diosmin is a phlebotropic drug frequently used in the management of chronic venous insufficiency (CVI). It prevents degradation of noradrenaline and improves venous tone, reduces capillary permeability and improves lymphatic drainage. We studied the effect of diosmin on post-CABG pedal edema and pedal edema associated with deep venous thrombosis. Twenty-five patients of chronic venous insufficiency and 20 patients with leg edema following CABG with saphenous vein graft were randomized to 450 mg thrice a day of diosmin or a placebo for a period of six weeks. Patients were followed up fortnightly to look for signs of chronic venous insufficiency. Symptoms of pain, night cramps, etc. were assessed on a scale of 10 on each follow-up. Edema was assessed by measuring leg circumference. Symptom score improved by 4.84±3.24 and 3.5±3.34 in the treatment groups of both chronic venous insufficiency and post-CABG patients. It improved by 3±2.56 and 2.5±2.67 in the placebo groups of both chronic venous insufficiency and post-CABG patients. The leg circumference improved by 3.61±1.85 and 4.15±2.41 cm in the treatment group and 0.94±1.18 and 1.1±2.81 cm in the placebo group among patients with CVI and post-CABG, respectively, and this improvement was statistically significant. There were no significant side-effects from the drug. Diosmin is effective in improving leg edema in patients with CVI and in patients who undergo coronary bypass using saphenous vein grafts.

*link no longer available*


PYCNOGENOL in chronic venous insufficiency.

Petrassi C, Mastromarino A, Spartera C.

Cattedra e Scuola di Specializzazione in Chirurgia Vascolare, Dipartimento di Scienze Chirurgiche, Universita degli Studi di L'Aquila, Italy.

The aim of out study was to investigate the efficacy of Pycnogenol - a French maritime pine bark extract - in the treatment of chronic venous insufficiency (CVI). The study consisted of a double-blind phase - in which 20 patients were recruited and randomly treated with placebo or Pycnogenol (100 mg 2 3/day for 2 months) - and an open phase - in which other 20 patients were treated with Pycnogenol at the same dose schedule. In total, 40 patients were enrolled; 30 of them were treated with Pycnogenol and 10 with placebo. Pycnogenol significantly improved the legs' heaviness and subcutaneous edema; the venous pressure was also significantly reduced by the Pycnogenol treatment, thus adding further clinical evidence to its therapeutic efficacy in patients with CVI. Pycnogenol was effective, probably by either stabilizing the collagenous subendothelial basal membrane or scavenging the free radicals, or by a combination of these activities. Clinically, capillary leakage, perivascular inflammation and subcutaneous edema were all reduced. The safety of use of Pycnogenol is demonstrated by the lack of side effects or changes in blood biochemistry and hematologic parameters. Pycnogenol can be therefore recommended both for prevention and treatment of CVI and related veno-capillary disturbances.

PMID: 11081989 |PubMed - indexed for MEDLINE]


External Links


Chronic Venous Insufficiency (1)



Chronic Venous Insufficiency


Chronic Venous Insufficiency and Postphlebitic Syndrome



Chronic Venous Insufficiency

Family Practice Notebook


Chronic venous insufficiency and leg ulceration



Venous insufficiency and ulceration: a review



Venous dynamics in leg lymphedema.

Filaria Journal


The aetiology and pathophysiology of chronic venous insufficiency and leg ulcers


Pathophysiology of chronic venous disease


Compresion Treatment for Chronic Venous Insufficiency


Endovascular treatment of patients with chronic cerebrospinal venous insufficiency and multiple sclerosis.  

Oct 2011 



Venoruton®: post thrombotic syndrome. Clinical improvement in venous insufficiency (signs and symptoms) with Venoruton®. A five-year, open-registry, efficacy study. 

Sept. 2011


Percutaneous management of chronic deep venous reflux: review of experimental work and early clinical experience with bioprosthetic valve.

Feb 2008

Sage Publications


Chronic venous insufficiency. Pathogenesis and modern diagnostic possibilities




Clinical and capillaroscopic evaluation in the treatment of chronic venous insufficiency with Ruscus aculeatus, hesperidin methylchalcone and ascorbic acid in venous insufficiency treatment of ambulatory patients.

Dec 2007



Effectiveness of mesoglycan in patients with previous deep venous thrombosis and chronic venous insufficiency

Dec 2007



ICD-10 and ICD-9 Codes


I87.2, Venous insufficiency (chronic)(peripheral

I87.8, Other specified disorders of veins

I87.9, Disorder of vein, unspecified


459.81 Venous insufficiency, unspec.

* additional code for any associated ulceration (707.10-707.9)


Caption: Picture 7. Lower leg venous anatomy.
Click to see larger picture


Caption: Picture 8. Perforating veins of the lower leg.
Click to see larger picture
Caption: Picture 3. Perforator vein bulging into subcutaneous tissue.
Click to see larger picture
Caption: Picture 5. Venous stasis ulcer and surrounding dystrophic tissue.
Click to see larger picture

e Medicine


See also:


Edema and Chronic Venous Insufficiency

Edema and Deep Venous Thrombosis

Edema and Reflex Sympathetic Dystrophy/Complex Regional Pain Syndrome

Edema and Venous Pooling


Edema of the Neck

Edema and Nephrotic Syndrome

Edema of the Face


Edema and Diabetes


Join us as we work for lymphedema patients everywehere:

Advocates for Lymphedema

Dedicated to be an advocacy group for lymphedema patients. Working towards education, legal reform, changing insurance practices, promoting research, reaching for a cure.


Pat O'Connor

Lymphedema People / Advocates for Lymphedema


For information about Lymphedema\

For Information about Lymphedema Complications

For Lymphedema Personal Stories

For information about How to Treat a Lymphedema Wound

For information about Lymphedema Treatment

For information about Exercises for Lymphedema

For information on Infections Associated with Lymphedema

For information on Lymphedema in Children

Lymphedema Glossary


Lymphedema People - Support Groups


Children with Lymphedema

The time has come for families, parents, caregivers to have a support group of their own. Support group for parents, families and caregivers of chilren with lymphedema. Sharing information on coping, diagnosis, treatment and prognosis. Sponsored by Lymphedema People.



Lipedema Lipodema Lipoedema

No matter how you spell it, this is another very little understood and totally frustrating conditions out there. This will be a support group for those suffering with lipedema/lipodema. A place for information, sharing experiences, exploring treatment options and coping.

Come join, be a part of the family!




If you are a man with lymphedema; a man with a loved one with lymphedema who you are trying to help and understand come join us and discover what it is to be the master instead of the sufferer of lymphedema.



All About Lymphangiectasia

Support group for parents, patients, children who suffer from all forms of lymphangiectasia. This condition is caused by dilation of the lymphatics. It can affect the intestinal tract, lungs and other critical body areas.



Lymphatic Disorders Support Group @ Yahoo Groups

While we have a number of support groups for lymphedema... there is nothing out there for other lymphatic disorders. Because we have one of the most comprehensive information sites on all lymphatic disorders, I thought perhaps, it is time that one be offered.


Information and support for rare and unusual disorders affecting the lymph system. Includes lymphangiomas, lymphatic malformations, telangiectasia, hennekam's syndrome, distichiasis, Figueroa
syndrome, ptosis syndrome, plus many more. Extensive database of information available through sister site Lymphedema People.



Lymphedema People New Wiki Pages

Have you seen our new “Wiki” pages yet?  Listed below are just a sample of the more than 140 pages now listed in our Wiki section. We are also working on hundred more.  Come and take a stroll! 

Lymphedema Glossary 


Arm Lymphedema 

Leg Lymphedema 

Acute Lymphedema 

The Lymphedema Diet 

Exercises for Lymphedema 

Diuretics are not for Lymphedema 

Lymphedema People Online Support Groups 



Lymphedema and Pain Management 

Manual Lymphatic Drainage (MLD) and Complex Decongestive Therapy (CDT) 

Infections Associated with Lymphedema 

How to Treat a Lymphedema Wound 

Fungal Infections Associated with Lymphedema 

Lymphedema in Children 


Magnetic Resonance Imaging 

Extraperitoneal para-aortic lymph node dissection (EPLND) 

Axillary node biopsy

Sentinel Node Biopsy

 Small Needle Biopsy - Fine Needle Aspiration 

Magnetic Resonance Imaging 

Lymphedema Gene FOXC2

 Lymphedema Gene VEGFC

 Lymphedema Gene SOX18

 Lymphedema and Pregnancy

Home page: Lymphedema People

Page Updated: Jan. 8, 2012