Venous Pooling
Edema and Venous Pooling
Lymphedema and Venous Pooling
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Edema and Venous Pooling
Related Terms: venous return, deep venous thrombosis, blood clot, venous stasis, chronic venous insufficiency, leg edema, syncope, orthostatic stress, vasoconstriction of arterioles, post phlebitic syndrome, post thrombotic syndrome, Postural Tachycardia Syndrome, venous leg ulcer, venous congestion, lymphedema, venous reflux
Discussion
Increase in blood contained in veins of lower limbs occurring on standing. Results in fall in pulse pressure and consequential baroreflex increase in heart rate and vasoconstriction of arterioles. Putting it in layman's terms it is simply a poor return of the blood from the legs and feet to the heart. One resulting complication is edema of the affected limb. The edema results from fluid and plasma collecting within the limb instead of processing through the cardiovascular system.
Venous pooling occurs due to lack of sympathetic activity and loss of active muscle function in the lower extremities and trunk musculature in persons with a spinal cord injury. This produces a smaller venous blood return to the heart, thus reducing stroke volume and cardiac output. To decrease this effect during exercise, an abdominal binder can be worn. This produces increased intra-abdominal pressure to help prevent redistribution of blood to the abdomen and lower extremities. This, in turn, helps prevent orthostatic hypotension (a sudden drop in systolic blood pressure greater than 20 mm Hg or a drop in diastolic blood pressure greater than 10 mm Hg resulting in nausea, dizziness, or fainting when in an upright position). The binder also helps with breathing.
Etiology
The main cause of this condition is the lack of, or damage to the bicuspid valves in the veins. In the normal pumping action of the heart blood flow is stopped from "backwashing" during the resting period of the heart from these valves. When damaged or when congenitally not there, the blood flows backwards. This can be either congenital or from trauma or disease.
Venous hypertension in diseased veins is thought to cause CVI by the following sequence of events. Increased venous pressure transcends the venules to the capillaries, impeding flow. Low-flow states within the capillaries cause leukocyte trapping. Trapped leukocytes release proteolytic enzymes and oxygen free radicals, which damage capillary basement membranes. Plasma proteins, such as fibrinogen, leak into the surrounding tissues, forming a fibrin cuff. Interstitial fibrin and resultant edema decrease oxygen delivery to the tissues, resulting in local hypoxia. Inflammation and tissue loss result. (1)
Another cause is Postural tachycardia syndrome (POTS). This is a disorder that is characterized by a pulse rate that is too high when the patient is standing. Instead of the blood returning it pools due to the inefficient pump action of the heart. Symptoms of this disorder include rapid heartbeat, lightheadedness with prolonged standing, headache, chronic fatigue, chest pain, and other nonspecific complaints.
Finally, prolonged sitting in cramped position will cause venous pooling. This is especially important to lymphedema patients. On any prolonged trip, patients with this condition should make every effort to move around, stand, stretch or walk.
Symptoms
The most obvious symptom is the swelling that will occur in the foot and lower leg of the affected limb. Other symptoms may include tight calves, legs and feet that feel "heavy," tired, achy or restless.
Treatment
Treatment for venous pooling will ultimately focus on the underlying condition that causes it.
For the edema involved, that patient may need decongestive massage therapy to gently move the fluids from the leg or foot. Compression wrappings or compression hosery may be needed as well.
In the condition of the congenital lack of vein valves or in the situation of damaged vascular veins, surgical grafts may be required.
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Abstracts and Studies
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Reflex
responses to regional venous pooling during lower body negative
pressure in
humans
Abstract
John R. Halliwill1, Lori A. Lawler1, Tamara J. Eickhoff1, Michael J.
Joyner1 and
Sharon L. Mulvagh2
1 Departments of Anesthesiology, Physiology, and Biophysics, Mayo
Medical
School, and 2 Division of Cardiovascular Diseases and Internal
Medicine, Mayo
Clinic and Foundation, Rochester, Minnesota 55905
http://jap.physiology.org/cgi/content/abstract/84/2/454
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Reflex responses to regional venous pooling during lower body
negative
pressure in humans
Abstract
http://www.uth.tmc.edu/apstracts/1997/jap/October/436A.html
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Syncope (Fainting) - Venous Pooling
http://www.txai.org/edu/syncope/mechanisms.htm
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Capillaries, Veins and Venules, & Lymphatics
http://www.iuvascular.com/Unthank/Teach/Quiz2Answers.html
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Blood Vessels and Venous Pooling
http://greenfield.fortunecity.com/rattler/46/veins.htm
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Chronic Venous Insufficiency (1)
E Medicine
http://www.emedicine.com/med/topic2760.htm
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The
image on the right shows a cutaway section through a large vein
The vessels have a relatively large diameter (the vena cava is 2-3 cm in diameter) and thus offer low resistance to blood flow. Some veins, especially in the arms and legs, have internal folds of the endothelial lining that function as valves and allow blood to flow in one direction only, towards the heart. These valves can be damaged if over stretched by high venous pressures for long periods, for example during pregnancy or in people who stand for extended periods; the valves become incompetent, lose their function, and varicose veins develop. As a result of this, oedema and varicose ulcers can develop.
A major part of the blood volume, approximately 60%, is contained within the venous system and for this reason veins are sometimes referred to as capacity vessels. The capacity of the venous system can be modified by altering the lumen size of the muscular venules and veins; the changes are mediated by altering the venomotor tone, that is, the degree of contraction of the smooth muscle in the tunica media. Venomotor tone is mainly under the control of the sympathetic nervous system. Changes in the venomotor tone can increase or decrease the capacity of the venous circulation and therefore can partially compensate for variations in the effective circulating blood volume.
http://greenfield.fortunecity.com/rattler/46/veins.htm
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http://www.nanomedicine.com/NMI/Figures/8.3.jpg
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See also:
Edema
http://www.lymphedemapeople.com/wiki/doku.php?id=edema
Edema and Chronic Venous Insufficiency
http://www.lymphedemapeople.com/thesite/edema_chronic_venous_insufficiency.htm
Edema and Deep Venous Thrombosis
http://www.lymphedemapeople.com/thesite/lymphedema_deep_venous_thrombosis.htm
Edema and Reflex Sympathetic Dystrophy/Complex Regional Pain Syndrome
http://www.lymphedemapeople.com/thesite/edema_and_reflex_sympathetic_dystrophy.htm
Edema and Venous Pooling
http://www.lymphedemapeople.com/thesite/edema_and_venous_pooling.htm
Angioedema
http://www.lymphedemapeople.com/wiki/doku.php?id=angioedema
Edema of the Neck
http://www.lymphedemapeople.com/thesite/lymphedema_of_the_neck.htm
Edema and Nephrotic Syndrome
http://www.lymphedemapeople.com/thesite/edema_nephrotic_syndrome.htm
Edema of the Face
http://www.lymphedemapeople.com/thesite/edema_of_the_face.htm
Thrombophlebitis
http://www.lymphedemapeople.com/wiki/doku.php?id=thrombophlebitis
Edema and Diabetes
http://www.lymphedemapeople.com/thesite/lymphedema_and_diabetes.htm
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